Inhibition of HtrA2/Omi ameliorates heart dysfunction following ischemia/reperfusion injury in rat heart in vivo

Immunology and Microbiology

• Reperfusion 83%
• High Temperature 50%
• Heart 50%
• Fas Ligand 33%
• Protein Degradation 33%
• Rat 33%
• Fas Receptor 16%
• Degradation 16%
• Apoptosis 16%
• Artery 16%
• Membrane 16%
• Mitochondrion 16%
• Cell Protection 16%
• Cytosol 16%
• Caspase 16%
• Mean Arterial Pressure 16%
• Reduction (Chemistry) 16%
• Processing 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Ischemia 50%
• Reperfusion Injury 50%
• Fas Ligand 33%
• Heart Disease 33%
• X Linked Inhibitor of Apoptosis 33%
• Rat 33%
• Caspase 3 16%
• Thiobarbituric Acid 16%
• Biphenyl 16%
• Infarction 16%
• Caspase 16%
• Cytoprotection 16%
• Serine Proteinase 16%
• Fodrin 16%
• Caspase 9 16%
• Inhibitor of Apoptosis Protein 16%
• Caspase 7 16%
• Apoptosis 16%

Neuroscience

• Ischemia 100%
• Reperfusion Injury 50%
• In Vivo 33%
• Fas Ligand 33%
• X Linked Inhibitor of Apoptosis 33%
• Apoptosis 16%
• Mitochondrion 16%
• Caspase 16%
• Caspase-9 16%
• Caspase 3 16%
• Serine Protease 16%
• Fodrin 16%
• Thiobarbituric Acid 16%
• Caspase 7 16%
• Inhibitor of Apoptosis Protein 16%