Growth inhibition by metabolites of γ-hexachlorocyclohexane in Sphingobium japonicum UT26

Ryo Endo, Yoshiyuki Ohtsubo, Masataka Tsuda, Yuji Nagata

研究成果: Article査読

13 被引用数 (Scopus)

抄録

The growth of a γ-hexachlorocyclohexane (γ-HCH)-degrading bacterium Sphingobium japonicum (formerly Sphingomonas paucimobilis) UT26 in rich medium was inhibited by γ-HCH. This growth inhibition was not observed in a mutant that lacked the initial or second step enzymatic activity for γ-HCH degradation, suggesting that metabolites of γ-HCH are toxic to UT26. Two metabolites of γ-HCH, 2,5-dichlorophenol (2,5-DCP) and 2,5-dichlorohydroquinone (2,5-DCHQ), showed a direct toxic effect on UT26 and other sphingomonad strains. Because only 2,5-DCP accumulated during γ-HCH degradation, 2,5-DCP is thought to be a main compound for growth inhibition.

本文言語English
ページ(範囲)1029-1032
ページ数4
ジャーナルBioscience, Biotechnology and Biochemistry
70
4
DOI
出版ステータスPublished - 2006

ASJC Scopus subject areas

  • バイオテクノロジー
  • 分析化学
  • 生化学
  • 応用微生物学とバイオテクノロジー
  • 分子生物学
  • 有機化学

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